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doi: 10.1016/j.trecan.2022.10.007 21 GoossensPRodriguez-VitaJEtzerodtAMasseMRastoinOGouirandVet al

Macrophages derived from Nox2-deficient (gp91phox/) mice produced marginal amounts of superoxide radical and are more susceptible to parasite infection than those macrophages derived from wild-type mice

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Homolytic bond fission can occur in vivo, leading to the production of free radicals

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From day 1 of the experiment to day 5 the escape latency was found to remarkably decrease, however, escape latency in the APP/PS1 mice was higher than that of the wild type mice